Author Topic: Fitness myths.  (Read 6431 times)

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Offline lonely moa

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Re: Fitness myths.
« Reply #75 on: Apr 29, 2012, 01:27:44 AM »


Fasted cardio or workouts: scientific studies say no,  there is no magic in fasted cardio that causes people to burn more fat.

Second point, it depends on you goals. Protein and carbs post workout will spike insulin which is anabolic.



Show me the studies please.  I am training my body to use a higher % of fat as fuel as opposed to glycogen because there are much greater stores of fat to draw upon.  There are a few pro triathletes who have used this method successfully.  I am not talking about 40min on the elliptical either.  Obviously you won't deplete glycogen stores in that short of a workout.  I am talking about a medium intensity, 4-5 hour ride on my bike or a 2-3 hour run.  I guarantee you that I will have burned through the 1000 or so calories of stored glycogen in my muscles by then and my body will have no choice but to start metabolizing fat for fuel.  I can tell you that it works.

http://triathlete-europe.competitor.com/2011/06/15/torbjorn-sindballe-how-to-turn-your-fat-into-fuel/


Have a look at http://www.bengreenfieldfitness.com/  He is one of the few triathaletes you speak of.  He does Kona and at the last one, he dropped the electrolytes.  His strategy is just as you describe.  He says he moderately carb loads for a few days for two or three events a year.  He has just publioshed a book, "Low Carbohydrate Diet For Triathletes".  Haven't read it but you could check it out.  I don't tri but I do play hard when I'm well.

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Offline Alex Simmons

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Re: Fitness myths.
« Reply #76 on: Apr 29, 2012, 06:43:22 AM »
This thread reminds me of the quote:

The plural of anecdote is not evidence.

Offline Tatyana

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Re: Fitness myths.
« Reply #77 on: Apr 29, 2012, 06:58:52 AM »


Fasted cardio or workouts: scientific studies say no,  there is no magic in fasted cardio that causes people to burn more fat.

Second point, it depends on you goals. Protein and carbs post workout will spike insulin which is anabolic.



Show me the studies please.  I am training my body to use a higher % of fat as fuel as opposed to glycogen because there are much greater stores of fat to draw upon.  There are a few pro triathletes who have used this method successfully.  I am not talking about 40min on the elliptical either.  Obviously you won't deplete glycogen stores in that short of a workout.  I am talking about a medium intensity, 4-5 hour ride on my bike or a 2-3 hour run.  I guarantee you that I will have burned through the 1000 or so calories of stored glycogen in my muscles by then and my body will have no choice but to start metabolizing fat for fuel.  I can tell you that it works.

http://triathlete-europe.competitor.com/2011/06/15/torbjorn-sindballe-how-to-turn-your-fat-into-fuel/


Despite people saying they can 'train' their metabolic pathways to switch which substrate it burns, I am fairly certain this is magical thinking with a wee bit of pseudoscience.

You could just as easily be burning muscle tissue as fat.

How are you testing yourself to know that you are switching substrates to fat?

I am only aware of one home method for this and there are some major issues with it, especially in trained athletes.

I also think that  there is some biochemical variability that may benefit some people, but it is not the 'magic bullet' and the 'fat burning rule' set in stone and just because you have read something and like how it sounds, it doesn't mean that it will apply to you.


I must admit I pinched this from Layne Norton.

Quote
A new article on fasted cardio appearing in the Strength and Conditioning Journal by Brad Schoenfeld, MSc, CSCS

I put the full text below for those who are interested. For those that are lazy to read the whole thing here are the cliffs:

-research has shown no difference in total fat loss between subjects doing fasted cardio and those doing cardio after eating.

-fat burning consists of 1) liberating fatty acids from adipose tissue through lipolysis and then transport of those fatty acids to other tissues like muscle, liver, heart where they are then 2) oxidized for energy. When you eat before cardio you reduce lipolysis but it ends up not making a difference because lipolysis is NOT the rate limiting step of fat loss when it comes to cardio, it is oxidation that is rate limiting so you end up oxidizing the same amount

-you may burn MORE fat over a 24 hour period when you eat beforehand because there is a GREATER thermogenic response to cardio as opposed to eating fasted

-Lemon et al. demonstrated nitrogen losses were DOUBLED when you train fasted. Fantastic for maintaining muscle in a caloric deficit... NOT

-not eating before cardio will reduce training intensity and means you will burn less calories during cardio because you won't have as much energy.

here is the full article

--------------------------------------------------------------------------------------

Does Cardio After an Overnight Fast Maximise Fat Loss?

A common fat burning strategy employed by bodybuilders, athletes, and fitness enthusiasts is to perform cardiovascular exercise early in the morning on an empty stomach. This strategy was popularized by Bill Phillips in his book, ‘‘Body for Life’’ (23). According to Phillips, performing 20 minutes of intense aerobic exercise after an overnight fast has greater effects on fat loss than performing an entire hour of cardio in the postprandial state. The rationale for the theory is that low glycogen levels cause your body to shift energy utilization away from carbohydrates, thereby allowing greater mobilization of stored fat for fuel. However, although the prospect of reducing the body fat by training in a fasted state may sound enticing, science does not support its efficacy.

First and foremost, it is shortsighted to look solely at how much fat is burned during an exercise session. The human body is very dynamic and continually adjusts its use of fat for fuel. Substrate utilization is governed by a host of factors (i.e., hormonal secretions, enzyme activity, transcription factors, etc), and these factors can change by the moment (27). Thus, fat burning must be considered over the course of days—not on an hour-to-hour basis—to get a meaningful perspective on its impact on body composition (13). As a general rule, if you burn more carbohydrate during a workout, you inevitably burn more fat in the post- exercise period and vice versa.
It should be noted that high-intensity interval training (HIIT) has proven to be a superior method for maximizing fat loss compared with a moderate- intensity steady-state training (10,26,29). Interestingly, studies show that blood flow to adipose tissue diminishes at higher levels of intensity (24). This is believed to entrap free fatty acids within fat cells, impeding their ability to be oxidized while training. Yet, despite lower fat oxidation rates during exercise, fat loss is nevertheless greater over time in those who engage in HIIT versus training in the ‘‘fat burning zone’’ (29), providing further evidence that 24-hour energy balance is the most important determinant in reducing body fat.

The concept of performing cardiovascular exercise on an empty stomach to enhance fat loss is flawed even when examining its impact on the amount of fat burned in the exercise session alone. True, multiple studies show that consumption of carbohydrate before low- intensity aerobic exercise (up to approximately 60% V_o2max) in untrained subjects reduces the entry of long-chain fatty acids in the mitochondria, thereby blunting fat oxidation (1,14,18,28). This is attributed to an insulin-mediated attenuation of adipose tissue lipolysis, an increased glycolytic flux, and a decreased expression of genes involved in fatty acid transport and oxidation (3,6,15). However, both training status and aerobic exercise intensity have been shown to mitigate the effects of a pre-exercise meal on fat oxidation (4,5,24). Recent research has shed light on the complexities of the subject.

Horowitz et al. (14) studied the fat burning response of 6 moderately trained individuals in a fed versus fasted state to different training intensities. Subjects cycled for 2 hours at varying intensities on 4 separate occasions. During 2 of the trials, they consumed a high-glycemic carbohydrate meal at 30, 60, and 90 minutes of training, once at a low intensity (25% peak oxygen consumption) and once at a moderate intensity (68% peak oxygen consumption). During the other 2 trials, subjects were kept fasted for 12–14 hours before exercise and for the duration of training. Results in the low-intensity trials showed that although lipolysis was suppressed by 22% in the fed state compared with the fasted state, fat oxidation remained similar between groups until 80–90 minutes of cycling. Only after this point was a greater fat oxidation rate observed in fasted subjects. Conversely, during moderate-intensity cycling, fat oxidation was not different between trials at any time—this is despite a 20–25% reduction in lipolysis and plasma Free fatty acid concentration.

More recently, Febbraio et al. (9) evaluated the effect of pre-exercise and during exercise carbohydrate consumption on fat oxidation. Using a crossover design, 7 endurance- trained subjects cycled for 120 minutes at approximately 63% of peak power output, followed by a ‘‘performance cycle’’ where subjects expended 7 kJ/(kg body weight) by pedaling as fast as possible. Trials were conducted on 4 separate occasions, with subjects given (a) a placebo before and during training, (b) a placebo 30 minutes before training and then a carbohydrate beverage every 15 minutes throughout exercise, (c) a carbohydrate beverage 30 minutes before training and then a placebo during exercise, or (d) a carbohydrate beverage both before and every 15 minutes during exercise. The study was carried out in a double- blind fashion with trials performed in random order. Consistent with previous research, results showed no evidence of impaired fat oxidation associated with consumption of carbohydrate either before or during exercise.

Taken together, these studies show that during moderate-to-high intensity cardiovascular exercise in a fasted state—and for endurance-trained individuals regardless of training intensity— significantly more fat is broken down than that the body can use for fuel. Free fatty acids that are not oxidized ultimately become re-esterified in adipose tissue, nullifying any lipolytic benefits afforded by pre-exercise fasting.

It should also be noted that consumption of food before training increases the thermic effect of exercise. Lee et al. (19) compared the lipolytic effects of an exercise bout in either a fasted state or after consumption of a glucose/milk (GM) beverage. In a crossover design, 4 experimental conditions were studied: low-intensity long duration exercise with GM, low-intensity long duration exercise without GM, high- intensity short duration exercise with GM, and high-intensity short duration exercise without GM. Subjects were 10 male college students who performed all 4 exercise bouts in random order on the same day. Results showed that ingestion of the GM beverage resulted in a significantly greater excess postexercise oxygen consumption compared with exercise performed in a fasted state in both high- and low- intensity bouts. Other studies have produced similar findings, indicating a clear thermogenic advantage associated with pre-exercise food intake (7,11).

The location of adipose tissue mobilized during training must also be taken into account here. During low-to- moderate intensity training performed at a steady state, the contribution of fat as a fuel source equates to approxi- mately 40–60% of total energy expen- diture (30). However, in untrained subjects, only about 50–70% of this fat is derived from plasma Free fatty acids; the balance comes from intra- muscular triglycerides (IMTG) (30).
IMTG are stored as lipid droplets in the sarcoplasm near the mitochondria (2), with the potential to provide approximately two-thirds the available energy of muscle glycogen (32). Similar to muscle glycogen, IMTG can only be oxidized locally within the muscle. It is estimated that IMTG stores are approximately 3 times greater in type I versus type II muscle fibers (8,21,31), and lipolysis of these stores are max- imally stimulated when exercising at 65%V_o2max(24).

The body increases IMTG stores with consistent endurance training, which results in a greater IMTG utilization for more experienced trainees (12,16,22,31). It is estimated that nonplasma fatty acid utilization during endurance exercise is approximately twice that for trained versus untrained individuals (24,32). Hurley et al. (17) reported that the contribution of IMTG stores in trained individuals equated to approximately 80% of the total body fat utilization during 120 minutes of moderate- intensity endurance training.
The important point here is that IMTG stores have no bearing on health and/or appearance; it is the subcutaneous fat stored in adipose tissue that influences body composition. Consequently, the actual fat burning effects of any fitness strategy intended to increase fat oxida- tion must be taken in the context of the specific adipose deposits providing energy during exercise.

Another factor that must be considered when training in a fasted state is its impact on proteolysis. Lemon and Mullin (20) found that nitrogen losses were more than doubled when training while glycogen depleted compared with glycogen loaded. This resulted in a protein loss estimated at 10.4% of the total caloric cost of exercise after 1hourofcyclingat61%V_o2max.This would suggest that performing cardio- vascular exercise while fasting might not be advisable for those seeking to maximize muscle mass.

Finally, the effect of fasting on energy levels during exercise ultimately has an effect on fat burning. Training early in the morning on an empty stomach makes it very difficult for an individual to train at even a moderate level of intensity. Attempting to engage in a HIIT style routine in a hypoglycemic state almost certainly will impair performance (33). Studies show that a pre-exercise meal allows an individual to train more intensely compared with exercise while fasting (25). The net result is that a greater number of calories are burned both during and after physicalactivity,heightening fat loss.

In conclusion, the literature does not support the efficacy of training early in the morning on an empty stomach as a tactic to reduce body fat. At best, the net effect on fat loss associated with such an approach will be no better than training after meal consumption, and quite possibly, it would produce inferior results. Moreover, given that training with depleted glycogen levels has been shown to increase proteolysis, the strategy has potential detrimental effects for those concerned with muscle strength and hypertrophy.

Offline LumpyFish

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Re: Fitness myths.
« Reply #78 on: Apr 29, 2012, 09:32:34 AM »
What about people who are in ketosis?

Offline Tatyana

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Re: Fitness myths.
« Reply #79 on: Apr 29, 2012, 09:59:55 AM »
What about people who are in ketosis?



As far as exercise and fitness goes, 'ketosis' is another buzz word associated with the whole low carb fad diets.

Most people are not applying the term ketosis in its proper biochemical, physiological or pathological context.

You would have to be more specific about what you mean by ketosis.

PS. How would you know if you are in ketosis or not?

« Last Edit: Apr 29, 2012, 10:14:14 AM by Tatyana »

Offline karirafn

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Re: Fitness myths.
« Reply #80 on: Apr 29, 2012, 11:22:20 AM »
I have no idea what Plastique means with that he's a "non-responder" given how the system works.
Well, it's difficult to grasp, but let me try to break it down. Ready? Here goes.

Creatine doesn't work for me.

Phew, that was tough. I can see how you'd be confused by "non-responder", though. Very ambiguous.

I realized that's what you think. I was wondering if you had an explanation for why you think that other than "it's just something I've noticed". Maybe I should have been clearer and asked it straight out. I actually didn't know that there was such a thing as a creatine nonresponder. So nice of you to feel the need to be an ass about it though.

There are all sorts of non-responders, this is a very well established pharmacological phenomena.

For example, for any given anti-depressant, only 40-60% of people will have a response.

ACE-inhibitors will not work on most people of African descent.

There are people who are codeine hyper metabolisers so there is absolutely no pain relieve benefit to them.

There are people who do not respond to acetylminophen (paracetamol) or to salicylate (aspirin). Seriously.

Some people do not respond to creatine.

As far as I can tell, I do, but I get so bored with taking supplements, I can't be bothered most of the time.

This would have been a proper response.

I did a little digging and found this study:

Quote
The results support a person-by-treatment interaction to acute Cr supplementation with R possessing a biological profile of lowest initial levels of Cr + PCr, greatest percentage of type II fibers, and greatest preload muscle fiber CSA and fat-free mass. Responders also showed improvement in 1RM leg press scores following the 5-day loading period. NR had higher preload levels of Cr + PCr, less type II muscle fibers, small preload muscle CSA, and lower fat-free mass and displayed no improvements in 1RM strength scores. The results suggest that to be considered a responder to acute oral supplementation, a favorable preexisting biological profile may determine the final extent to which an individual responds to supplementation. Physiologic profiles of nonresponders appear to be different and may limit their ability to uptake Cr. This may help partially explain the reported equivocal performance findings in the Cr supplementation literature.

(R = Responder, NR = Nonresponder, CSA = Cross-sectional area (biopsy of muscle fiber))

This thread reminds me of the quote:

The plural of anecdote is not evidence.

My thoughts exactly.
« Last Edit: Apr 29, 2012, 11:26:14 AM by karirafn »
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Offline lonely moa

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Re: Fitness myths.
« Reply #81 on: Apr 29, 2012, 01:15:11 PM »

PS. How would you know if you are in ketosis or not?

Testing one's urine with keto strips seems to work, although in healthy, active people not many ketones make it into the urine.  Many tissues seem to prefer ketones for energy.

Keeping one's carbohydrate intake to less than a few tens of grams/day will work.  Belonging to any number of populations (before the introduction of carbohydrates to thd diet) like Inuit, Masai or a South Island Maori would qualify.  Reading the accounts of explorers and settlers would lead one to believe that they must have been in ketosis for years.
“Most people would rather die than think; in fact, they do so”

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Offline Tatyana

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Re: Fitness myths.
« Reply #82 on: Apr 29, 2012, 03:39:27 PM »

PS. How would you know if you are in ketosis or not?

Testing one's urine with keto strips seems to work, although in healthy, active people not many ketones make it into the urine.  Many tissues seem to prefer ketones for energy.

Keeping one's carbohydrate intake to less than a few tens of grams/day will work.  Belonging to any number of populations (before the introduction of carbohydrates to thd diet) like Inuit, Masai or a South Island Maori would qualify.  Reading the accounts of explorers and settlers would lead one to believe that they must have been in ketosis for years.

Ketones in urine is contrary to the idea of 'substrate switching' means that the body will be using the ketone bodies as a source of fuel.

If they are in your urine, it means you are probably metabolising muscle.

As well, the body will still be making glucose via the process of gluconeogenesis, anywhere from 20-120 g per day, depending on the diet as there are a number of tissues and cells that either prefer or NEED glucose, for example red blood cells need glucose.


Offline Plastique

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Re: Fitness myths.
« Reply #83 on: Apr 29, 2012, 03:44:42 PM »
I realized that's what you think. I was wondering if you had an explanation for why you think that other than "it's just something I've noticed".

No you weren't, you were superciliously casting doubt on my statement because you thought it wasn't possible to be a non-responder based on how creatine affects muscle power.

And you do it again with "I realize that's what you think", and by assuming it's "something I've noticed" as if I have no authority to assert my response to creatine unless I can cite a double-blind placebo-controlled study to support it. Your shitty little "my thoughts exactly" reply to Alex's post about anecdote emphasizes this.

Offline jt512

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Re: Fitness myths.
« Reply #84 on: Apr 29, 2012, 04:45:35 PM »
[N]trition is a major component of body composition . . .

Sounds like a fitness myth itself.

Jay

Offline LumpyFish

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Re: Fitness myths.
« Reply #85 on: Apr 29, 2012, 05:54:42 PM »
What about people who are in ketosis?



As far as exercise and fitness goes, 'ketosis' is another buzz word associated with the whole low carb fad diets.

Most people are not applying the term ketosis in its proper biochemical, physiological or pathological context.

You would have to be more specific about what you mean by ketosis.

PS. How would you know if you are in ketosis or not?

I was just under the impression that you can tell if you are in ketosis by testing urine with some sort of strips.    And I heard, though you would be much better to inform me, that ketones are just the by product of fat metabolism. So even if you are not doing low carb, but are losing weight/fat, there will be at least trace ketones in your urine

Offline LumpyFish

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Re: Fitness myths.
« Reply #86 on: Apr 29, 2012, 05:56:05 PM »

PS. How would you know if you are in ketosis or not?

Testing one's urine with keto strips seems to work, although in healthy, active people not many ketones make it into the urine.  Many tissues seem to prefer ketones for energy.

Keeping one's carbohydrate intake to less than a few tens of grams/day will work.  Belonging to any number of populations (before the introduction of carbohydrates to thd diet) like Inuit, Masai or a South Island Maori would qualify.  Reading the accounts of explorers and settlers would lead one to believe that they must have been in ketosis for years.

Ketones in urine is contrary to the idea of 'substrate switching' means that the body will be using the ketone bodies as a source of fuel.

If they are in your urine, it means you are probably metabolising muscle.

As well, the body will still be making glucose via the process of gluconeogenesis, anywhere from 20-120 g per day, depending on the diet as there are a number of tissues and cells that either prefer or NEED glucose, for example red blood cells need glucose.
Does this occur even if one is eating a sufficient number of calories and grams of protein?

Offline karirafn

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Re: Fitness myths.
« Reply #87 on: Apr 29, 2012, 05:58:13 PM »
I realized that's what you think. I was wondering if you had an explanation for why you think that other than "it's just something I've noticed".
No you weren't, you were superciliously casting doubt on my statement because you thought it wasn't possible to be a non-responder based on how creatine affects muscle power.
Yes I was, that doesn't change what I said in my last comment. Since I had never heard of that term before and you gave no explanation of it I don't think my comment was that outrageous. I assumed the explanation for your claim might be something along the lines of GodSlayer's comment:
or you store heaps and never exhaust it? or, the difference is just not all that obvious (observation bias/placebo effect in other users?)
I know I should have just googled the term before making the comment but I didn't so that one is on me.
And you do it again with "I realize that's what you think", and by assuming it's "something I've noticed" as if I have no authority to assert my response to creatine unless I can cite a double-blind placebo-controlled study to support it.
Why should I assume that when you gave no additional information? I never asked for a double-blind placebo-controlled study.

I'm not interested in arguing about such a trivial matter so I consider it closed.
Your shitty little "my thoughts exactly" reply to Alex's post about anecdote emphasizes this.
My shitty little reply to Alex's post was not directed at you. I was simply agreeing with him that there are many anecdotal stories in the thread that people are using to support their argument.
« Last Edit: Apr 29, 2012, 06:06:12 PM by karirafn »
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Offline Tatyana

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Re: Fitness myths.
« Reply #88 on: Apr 29, 2012, 06:31:20 PM »
What about people who are in ketosis?



As far as exercise and fitness goes, 'ketosis' is another buzz word associated with the whole low carb fad diets.

Most people are not applying the term ketosis in its proper biochemical, physiological or pathological context.

You would have to be more specific about what you mean by ketosis.

PS. How would you know if you are in ketosis or not?

I was just under the impression that you can tell if you are in ketosis by testing urine with some sort of strips.    And I heard, though you would be much better to inform me, that ketones are just the by product of fat metabolism. So even if you are not doing low carb, but are losing weight/fat, there will be at least trace ketones in your urine

No.

If there are a lot of ketone bodies in your urine, you are probably a diabetic in ketoacidosis.

The idea of generating ketone bodies is that the body will use the ketone bodies (which are a byproduct of beta-oxidation of fatty acids), not excrete them as waste.

I have done low carb diets and had the ketone testing strips. People who do this are typically highly disappointed at the lack of any sort of trace ketones in the urine.

It just doesn't happen.

Offline Tatyana

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Re: Fitness myths.
« Reply #89 on: Apr 29, 2012, 06:36:20 PM »

PS. How would you know if you are in ketosis or not?

Testing one's urine with keto strips seems to work, although in healthy, active people not many ketones make it into the urine.  Many tissues seem to prefer ketones for energy.

Keeping one's carbohydrate intake to less than a few tens of grams/day will work.  Belonging to any number of populations (before the introduction of carbohydrates to thd diet) like Inuit, Masai or a South Island Maori would qualify.  Reading the accounts of explorers and settlers would lead one to believe that they must have been in ketosis for years.

Ketones in urine is contrary to the idea of 'substrate switching' means that the body will be using the ketone bodies as a source of fuel.

If they are in your urine, it means you are probably metabolising muscle.

As well, the body will still be making glucose via the process of gluconeogenesis, anywhere from 20-120 g per day, depending on the diet as there are a number of tissues and cells that either prefer or NEED glucose, for example red blood cells need glucose.
Does this occur even if one is eating a sufficient number of calories and grams of protein?

Do you  mean gluconeogenesis? It occurs all the time. Our metabolism is not like this on/off switch where we suddenly turn on insulin and turn on glycolysis and turn off beta-oxidation.

It is more like a dimmer switch where some glycolysis is occurring while beta oxidation is occuring and a wee bit of gluconeogenesis is happening.

For example, most people do not realise that we have a background level of insulin present at all times, and that our pancreas releases insulin every twenty minutes whether or not we eat or not.



Do you mean the breakdown of muscle tissue? It depends on a lot of factors and there is a huge amount of biochemical variation and bro-ology associated with the issue of muscle catabolism when doing low carb/fasted dieting.

It does appear that protein is muscle sparing.